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Evaluation test #11987/2017

The inflammation of the tumoral process is consecutive to:
There is no typical inflammation in this tumor.
It is as a consequence of the collision with an oral plasmacytoma.
Erosion and / or ulceration of the gingival epithelium.
A gingival hyperplasia secondary to the acanthomatous epulis.

The origin of the acanthomatous ameloblastoma is:
The enamel layer formed by the ameloblasts.
Periodontal ligament.
Gingival epithelium.
The enamel layer formed by the odontoblasts.

The prognosis of acanthomatous ameloblastoma is:
Poor prognosis due to invasion of lymphatic vessels.
Favorable although it can metastasize to regional lymph node.
Favorable because it has an expansive character.
Reserved for the infiltrative behaviour of this neoplastic process.

The distribution of the cells in the acanthomatous ameloblastoma is:
The cells are rounded, with scarce basophilic cytoplasm and that expand in sheats.
In cords that surround little cellular areas.
In nests that contain keratin inside.
The growth-pattern of spindle-shaped cells is characteristic.

The differential diagnosis of well localized gingival masses includes:
Whether mass is ulcerated it is an adenoma.
The gingival masses are exclusively inflammatory processes, not tumor.
Gingival hyperplasia, squamous cell carcinoma and acanthomatous ameloblastoma.
Squamous cell carcinoma and fibroma.